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Metabolic re-wiring that limits the malate-aspartate shuttle and mitochondrial substrate oxidation, and promotes aerobic glycolysis, the re-oxidation of NADH by MDH1, folate cycle NADPH production, and pentose phosphate supply by the non-oxidative PPP may thus constitute a successful strategy for minimizing ROS production and supporting high proliferation rates in oxygen-rich environments

the effect must be considered in the context of severe LPO (a 10- to 30-fold increase in LPO parameters)

Other experiments were conducted using fresh samples

they are a normal part of life and are produced by your dogs metabolism